β-Nicotinamide Adenine Dinucleotide NAD CAS 53-84-9 Powder Purity 99% Anti Aging

Product Description:

Product Name: β-Nicotinamide Adenine Dinucleotide NAD CAS NO 53-84-9

Synonyms:

beta-Diphosphopyridine nucleotide;

Coenzyme I Hydrate,oxidized form;

beta-NAD+;

Chemical & Physical Properties:

Appearance : White to off-white powder.

Assay : ≥99.0%

Melting point: 140-142℃

Solubility: Soluble in water, insoluble in ace tone and other organic solvents.

Stability: Stable. Hygroscopic. Incompatible with strong oxidizing agents.

Water Solubility: 50 mg/mL.

Application:

Niacinamide adenine dinucleotide is involved in various physiological activities such as cellular material metabolism, energy synthesis, and cellular DNA repair, and plays an important role in the body's immune system. In a healthy state, the concentration of nicotinamide adenine dinucleotide in the human body remains stable, maintaining normal cellular functions.

The concentration of nicotinamide adenine dinucleotide in the body determines the process and degree of cellular aging, and a decrease in concentration will accelerate the process of cellular aging.

Research has shown that NAD+has a protective effect on renal infarction caused by ischemic surgery, significantly reducing serum levels of urea nitrogen and creatinine; NAD+has a protective effect on renal tubular injury caused by ischemic surgery.

NAD+(Nicotinamide adenine dinucleotide, abbreviated as NAD+) can effectively protect against kidney injury caused by renal ischemia. NAD+has important application value in the preparation of drugs for the prevention and treatment of renal ischemic injury.

In addition, nicotinamide adenine dinucleotide has certain applications in the preparation of drugs for treating inflammatory pain. NAD+participates in regulating formalin and complete Freund's adjuvant (CFA) induced inflammatory pain through NAD+dependent deacetylases SIRT1 and SIRT2, while SIRT1 and SIRT2 participate in the inhibitory effect of NAD+on inflammatory pain through different mechanisms, thereby achieving analgesic effects on inflammatory pain.